Sabrina Carvalho Gomes* and Patricia Daniela Melchiors Angst
The supragingival plaque control need has been sustained by the concept of the periodontal disease as an opportunistic infection. This concept relies on the fact that the supragingival biofilm has been proven to be essential for the establishment of the subgingival biofilm, this opportunity arising through a retro-feeding process between the supragingival biofilm formation and maturation and the inflammatory response. Since decades, the impact of the supragingival plaque control has been recognized as an important factor to modulate the subgingival response, regarding clinical, microbiological, and immunological indicators. These studies collectively underscore the supragingival control as essential not only for prevention of periodontal disease and maintenance of therapy results but also for the treatment of periodontitis, favoring subgingival inflammation reduction as well as the stability of attachment levels. Thus, it can be concluded that an adequate supragingival control is essential for the quality of periodontal therapy, migrating from a second-plan intervention to a unique unquestionable importance.
Michel Goldberg* and Azumi Hirata
Derived from neural crests, the mature pulp is due to the proliferation and condensation of apical cells implicated in root lengthening. Adult cells are bound together by intercellular junctions (desmosomes and gap junctions), forming a network. They are further transported to the crown. Root lengthening is associated to tooth eruption and to vertical sliding. Composed by type I and III collagens, fibronectin, tenascin, and other non-collagenous proteins that include a series of proteoglycans, the extracellular matrix is favoring the sliding of pulp cells. A micro-vascular network differing in the root and crown, supply the blood flow. Endothelial cells, pericytes and lymphatic vessels are contributing to direct the vascular implication to pulp development. Inflammatory and immunocompetent cells are present with in the pulp, namely dendritic cells, macrophages, lymphocytes and endothelial cells. They add to the different forms of the immune response of the dental pulp implicated in the different types of programed cell death. Nerves play role in the neutrophin family, identified within the pulp. Neuromediators are released near the nerves. Stem cells issued from the pulp of permanent and deciduous teeth, from the apical papilla and from the dental follicle, contribute to pulp healing and regeneration. Sliding of the pulp cells from the apical to the coronal pulp, and later the lateral sliding due to the effects of Wnt visualized after BIO implantation, are factors implicated in the formation of reparative and reactionary dentin, and consequently to pulp healing.
Masae Ishihara, Yuji Sato, Noboru Kitagawa, Momoe Nakatsu*, Kana Takeda, Takuya Kakuda, Mari Takayama, Kensuke Tsubakida
Purpose: We aimed to clarify the effect of different measurement sites on the retention of maxillary complete dentures and to determine the optimal site and loading method for evaluating retention. The factors associated with mandibular complete denture retention are unclear; thus, a retention evaluation method has not been established. We investigated measurement methods and the optimal site by using a model to establish a retention measurement method for mandibular complete dentures.
Method: We manufactured complete resin dentures, based on normal methods used for edentulous mandibular models. We fabricated a covering splint to measure the denture surface. We set four points (A–D) for the measurement sites. We measured retention after filling the space between the denture mucosal surface and the model with four types of intervening saliva.
Results: All four types of intervening saliva could be measured only when measuring site A traction and the downward pressure on the fenestrations. Both conditions had a strong positive correlation (r = 0.94, P<.01). At site A, there was no significant difference in the measurement values with pressure exerted at45°and at 23°, which indicated a positive correlation (r = 0.73, P<.01).
Conclusion: Mandibular complete denture retention can be measured by applying oblique downward pressure on the occlusal plane at the mandibular central incisor midpoint.